The amount of chrysin in honey from various plant sources is about 0.2 mg per 100 g.[8] Chrysin is typically found at higher amounts in propolis than in honey.[9] A 2010 study found the amount of chrysin was 0.10 mg/kg in honeydew honey, and 5.3 mg/kg in forest honeys.[10] A 2010 study found the amount of chrysin in propolis was as much as 28 g/L.[10] A 2013 study found the amount of chrysin in various mushrooms from the island of Lesvos, Greece, varied between 0.17 mg/kg in Lactarius deliciosus to 0.34 mg/kg in Suillus bellinii.[10]
Bioavailability
The effects of chrysin are reliant on its bioavailability and solubility.[10] Following oral intake by humans, chrysin has low bioavailability and rapid excretion.[1] As a result, it is poorly absorbed.[1]
A 1998 study determined that the highest amounts in plasma was from 12 to 64 nM.[10] As of 2015, the serum levels of chrysin have not been cited in the literature.[10] Following oral intake by humans, the bioavailability was reported to be from 0.003% to 0.02%.[10]
Oral and topical application
There is insufficient information to determine how long chrysin has been used in pharmacy compounding.[11] Chrysin is used as an ingredient in dietary supplements, but there is no information on systemic exposure from topical application.[3] As of 2016, there was no evidence to support any effect of oral chrysin on testosterone levels,[4] or an any disease-modifying activity with oral or topical formulations.[12]
Safety
A daily consumed amount of chrysin of 0.5 to 3 g is considered safe.[6] As of 2016, there was no toxicity attributable to chrysin in clinical trials or adverse event reporting.[13] As of 2016, clinical safety issues have not been identified.[14] As of 2016, nonclinical data suggest potential concerns.[14] In 2016, the US Food and Drug Administration did not recommend chrysin be included on the list of bulk drug substances that can be used in compounding under section 503A of the Federal Food, Drug, and Cosmetic Act based on consideration of the following criteria: (1) physicochemical characterization; (2) safety; (3) effectiveness; and (4) historical use of the substance in compounding.[5]
Research
As of 2016, there is no evidence for chrysin being used in human clinical applications.[1] Research showed that orally administered chrysin does not have clinical activity as an aromatase inhibitor.[1][15]Nanoformulations of polyphenols, including chrysin, are made using various carrier methods, such as liposomes and nanocapsules.[16]
Bibliography
Brave M (23 June 2016). "Chrysin"(PDF). Pharmacy Compounding Advisory Committee, Division of Oncology Products, US Food and Drug Administration. pp. 1–13. This article incorporates text from this source, which is in the public domain.
^ abcdefgNabavi SF, Braidy N, Habtemariam S, Orhan IE, Daglia M, Manayi A, Gortzi O, Nabavi SM (2015). "Neuroprotective effects of chrysin: From chemistry to medicine". Neurochemistry International. 90: 224–31. doi:10.1016/j.neuint.2015.09.006. PMID26386393. S2CID24391203.
^Saarinen N, Joshi SC, Ahotupa M, Li X, Ammälä J, Mäkelä S, Santti R (September 2001). "No evidence for the in vivo activity of aromatase-inhibiting flavonoids". The Journal of Steroid Biochemistry and Molecular Biology. 78 (3): 231–9. doi:10.1016/S0960-0760(01)00098-X. PMID11595503. S2CID25787862.