Elspeth McLachlanFAA (born 22 July 1942), an Australian neuroscientist, is a world authority on neural pathways within the autonomic nervous system.[1] Her work has included detailed analyses of transmission in autonomic ganglia to studies of the organisation of autonomic nervous pathways and their disorder in pathological states, particularly injuries to peripheral nerves and the spinal cord.[2]
McLachlan was educated at the University of Sydney, from which institution she received Bachelor of Science degree in 1962, and a PhD in 1973, and a Doctor of Science degree in 1994.[3]
1985–1993 – Visiting Professor, Christian-Albrechts-Universität zu Kiel, Germany[3]
1987–1988 – National Health and Medical Research Council Principal Research Fellow and Associate Professor, School of Physiology and Pharmacology, University of New South Wales, Australia
1988–1993 – Professor and Head, Department of Physiology & Pharmacology, University of Queensland, Australia
1993–2011 – Senior Principal Research Fellow, Prince of Wales Medical Research Institute, Randwick, Australia
1999–2001 – Executive Head, Centre for Research Management, National Health & Medical Research Council, Canberra, Australia
2000–2003 – Investigador Asociado, Instituto de Neurociencias, Universidad Miguel Hernández, Alicante, Spain
2001–2006 – Pro-Vice Chancellor (Research) and Research Professor, University of New South Wales, Australia
2007–present – Emeritus Professor, University of New South Wales, Australia
Jänig, W.; McLachlan, E.M. (1992). "Characteristics of function-specific pathways in the sympathetic nervous system". Trends in Neurosciences. 15 (12): 475–481. doi:10.1016/0166-2236(92)90092-M. PMID1282743. S2CID9881261.
Sah, P; McLachlan, EM (1991). "Ca(2+)-activated K+ currents underlying the afterhyperpolarization in guinea pig vagal neurons: a role for Ca(2+)-activated Ca2+ release". Neuron. 7 (2): 257–64. doi:10.1016/0896-6273(91)90264-z. PMID1873029. S2CID9753479.
Tripovic, D., Pianova, S., McLachlan, E.M. and Brock, J.A. (2011) Slow and incomplete sympathetic reinnervation of rat tail artery restores the amplitude of nerve-evoked contractions provided a perivascular plexus is present. AJP Heart and Circulatory Physiology 300, H541-554