Most approved monoclonal antibodies are of the IgG1isotype, where two N-linked biantennary complex-type oligosaccharides are bound to the Fc region. The Fc region exercises the effector function of ADCC through its interaction with leukocyte receptors of the FcγR family. ADCC is important in the efficacy of cancer antibodies, but with many approved cancer antibodies there is less ADCC than could be desired due to nonspecific IgG competing with the drugs for binding to FcγIIIa on natural killer cells. Afucosylated monoclonal antibodies overcome this problem through improved FcγIIIa binding.[1]
Approaches
The Swiss company GlycArt Biotechnology developed a system using CHO cells, where the cells were engineered to overexpress an enzyme called GnTIII. The effect of this overexpression is to block the formation of fucosylated oligosaccharides on the expressed antibodies. This technology was first reported in 1999 and was the basis of GlycArt Biotechnology.[2]
Kyowa Hakko Kirin's "Potelligent" platform uses a CHO cell line in which FUT8 has been knocked out, and which produces antibodies with little to no fucose in the Fc region. The company gained marketing approval in Japan in April 2012 for a monoclonal antibody drug called mogamulizumab which was developed using the platform.[6] The Company's technology was first reported in 2004.[7]
Applications of afucosylated antibodies
Afucosylated antibodies are intensely used in the field of advanced medicine, also due to their high ADCC (antibody-dependent cellular cytotoxicity). This makes them effective in binding to specific targets while minimizing damage to surrounding tissue.
Some of the fields in which afucosylated antibodies are used or considered for application are:
cancer immunotherapy
autoimmune diseases
Infectious diseases
Furthermore, afucosylated antibodies are used as diagnostic tools and play a role in the development of personalized medications.[8]
References
^Satoh M, Iida S, Shitara K (2006). "Non-fucosylated therapeutic antibodies as next-generation therapeutic antibodies". Expert Opin Biol Ther. 6 (11): 1161–73. doi:10.1517/14712598.6.11.1161. PMID17049014. S2CID24803037.
^Umaña P, Jean-Mairet J, Moudry R, Amstutz H, Bailey JE (February 17, 1999). "Engineered glycoforms of an antineuroblastoma IgG1 with optimized antibody-dependent cellular cytotoxic activity". Nat. Biotechnol. 17 (2): 176–80. doi:10.1038/6179. PMID10052355. S2CID20078393.
^Yamane-Ohnuki N, Kinoshita S, Inoue-Urakubo M, Kusunoki M, Iida S, Nakano R, Wakitani M, Niwa R, Sakurada M, Uchida K, Shitara K, Satoh M (September 5, 2004). "Establishment of FUT8 knockout Chinese hamster ovary cells: an ideal host cell line for producing completely defucosylated antibodies with enhanced antibody-dependent cellular cytotoxicity". Biotechnol. Bioeng. 87 (5): 614–22. doi:10.1002/bit.20151. PMID15352059. S2CID40137283.